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Thomas J. Jentsch
Thomas J. Jentsch
Professor; FMP, MDC and Charité Berlin
在 fmp-berlin.de 的电子邮件经过验证 - 首页
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引用次数
引用次数
年份
Molecular structure and physiological function of chloride channels
TJ Jentsch, V Stein, F Weinreich, AA Zdebik
Physiological reviews 82 (2), 503-568, 2002
16912002
A potassium channel mutation in neonatal human epilepsy
C Biervert, BC Schroeder, C Kubisch, SF Berkovic, P Propping, ...
Science 279 (5349), 403-406, 1998
13881998
Loss of the ClC-7 chloride channel leads to osteopetrosis in mice and man
U Kornak, D Kasper, MR Bösl, E Kaiser, M Schweizer, A Schulz, ...
Cell 104 (2), 205-215, 2001
12322001
KCNQ4, a novel potassium channel expressed in sensory outer hair cells, is mutated in dominant deafness
C Kubisch, BC Schroeder, T Friedrich, B Lütjohann, A El-Amraoui, ...
Cell 96 (3), 437-446, 1999
10941999
Neuronal KCNQ potassium channels: physislogy and role in disease
TJ Jentsch
Nature Reviews Neuroscience 1 (1), 21-30, 2000
10002000
The skeletal muscle chloride channel in dominant and recessive human myotonia
MC Koch, K Steinmeyer, C Lorenz, K Ricker, F Wolf, M Otto, B Zoll, ...
Science 257 (5071), 797-800, 1992
8421992
A common molecular basis for three inherited kidney stone diseases
SE Lloyd, SHS Pearce, SE Fisher, K Steinmeyer, B Schwappach, ...
Nature 379 (6564), 445-449, 1996
8341996
Barttin is a Cl- channel β-subunit crucial for renal Cl- reabsorption and inner ear K+ secretion
R Estévez, T Boettger, V Stein, R Birkenhäger, E Otto, F Hildebrandt, ...
Nature 414 (6863), 558-561, 2001
7012001
Disruption of KCC2 reveals an essential role of K-Cl cotransport already in early synaptic inhibition
CA Hübner, V Stein, I Hermans-Borgmeyer, T Meyer, K Ballanyi, ...
Neuron 30 (2), 515-524, 2001
6862001
A chloride channel widely expressed in epithelial and non-epithelial cells
A Thiemann, S Gründer, M Pusch, TJ Jentsch
Nature 356 (6364), 57-60, 1992
6861992
Primary structure of Torpedo marmorata chloride channel isolated by expression cloning in Xenopus oocytes
TJ Jentsch, K Steinmeyer, G Schwarz
Nature 348 (6301), 510-514, 1990
6761990
Moderate loss of function of cyclic-AMP-modulated KCNQ2/KCNQ3 K+ channels causes epilepsy
BC Schroeder, C Kubisch, V Stein, TJ Jentsch
Nature 396 (6712), 687-690, 1998
6531998
ClC-5 Cl--channel disruption impairs endocytosis in a mouse model for Dent's disease
N Piwon, W Günther, M Schwake, MR Bösl, TJ Jentsch
Nature 408 (6810), 369-373, 2000
6292000
A constitutively open potassium channel formed by KCNQ1 and KCNE3
BÈC Schroeder, S Waldegger, S Fehr, M Bleich, R Warth, R Greger, ...
Nature 403 (6766), 196-199, 2000
6262000
Identification of LRRC8 heteromers as an essential component of the volume-regulated anion channel VRAC
FK Voss, F Ullrich, J Münch, K Lazarow, D Lutter, N Mah, ...
Science 344 (6184), 634-638, 2014
6122014
Disruption of ClC-3, a chloride channel expressed on synaptic vesicles, leads to a loss of the hippocampus
SM Stobrawa, T Breiderhoff, S Takamori, D Engel, M Schweizer, ...
Neuron 29 (1), 185-196, 2001
5972001
Voltage-dependent electrogenic chloride/proton exchange by endosomal CLC proteins
O Scheel, AA Zdebik, S Lourdel, TJ Jentsch
Nature 436 (7049), 424-427, 2005
5692005
Primary structure and functional expression of a developmentally regulated skeletal muscle chloride channel
K Steinmeyer, C Ortland, TJ Jentsch
Nature 354 (6351), 301-304, 1991
5181991
KCNQ4, a K+ channel mutated in a form of dominant deafness, is expressed in the inner ear and the central auditory pathway
T Kharkovets, JP Hardelin, S Safieddine, M Schweizer, A El-Amraoui, ...
Proceedings of the National Academy of Sciences 97 (8), 4333-4338, 2000
5152000
ClC-5, the chloride channel mutated in Dent’s disease, colocalizes with the proton pump in endocytotically active kidney cells
W Günther, A Lüchow, F Cluzeaud, A Vandewalle, TJ Jentsch
Proceedings of the National Academy of Sciences 95 (14), 8075-8080, 1998
5141998
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