Các bài viết có thể truy cập công khai - Georgia VelimeziTìm hiểu thêm
Có tại một số nơi: 11
DNA replication stress: causes, resolution and disease
A Mazouzi, G Velimezi, JI Loizou
Experimental cell research 329 (1), 85-93, 2014
Các cơ quan ủy nhiệm: Austrian Science Fund
Functional interplay between the DNA-damage-response kinase ATM and ARF tumour suppressor protein in human cancer
G Velimezi, M Liontos, K Vougas, T Roumeliotis, J Bartkova, M Sideridou, ...
Nature cell biology 15 (8), 967-977, 2013
Các cơ quan ủy nhiệm: Danish National Research Foundation
The DNA damage checkpoint precedes activation of ARF in response to escalating oncogenic stress during tumorigenesis
K Evangelou, J Bartkova, A Kotsinas, IS Pateras, M Liontos, G Velimezi, ...
Cell Death & Differentiation 20 (11), 1485-1497, 2013
Các cơ quan ủy nhiệm: US National Institutes of Health, Danish National Research Foundation
Modulation of the E2F1-driven cancer cell fate by the DNA damage response machinery and potential novel E2F1 targets in osteosarcomas
M Liontos, K Niforou, G Velimezi, K Vougas, K Evangelou, ...
The American journal of pathology 175 (1), 376-391, 2009
Các cơ quan ủy nhiệm: US National Institutes of Health
Map of synthetic rescue interactions for the Fanconi anemia DNA repair pathway identifies USP48
G Velimezi, L Robinson-Garcia, F Muñoz-Martínez, WW Wiegant, ...
Nature communications 9 (1), 2280, 2018
Các cơ quan ủy nhiệm: US National Institutes of Health, Austrian Science Fund, Cancer Research UK …
Parallel genome-wide screens identify synthetic viable interactions between the BLM helicase complex and Fanconi anemia
M Moder, G Velimezi, M Owusu, A Mazouzi, M Wiedner, ...
Nature communications 8 (1), 1238, 2017
Các cơ quan ủy nhiệm: US National Institutes of Health, Austrian Science Fund, Cancer Research UK
ATMIN is required for the ATM-mediated signaling and recruitment of 53BP1 to DNA damage sites upon replication stress
L Schmidt, M Wiedner, G Velimezi, J Prochazkova, M Owusu, S Bauer, ...
DNA repair 24, 122-130, 2014
Các cơ quan ủy nhiệm: Austrian Science Fund
DNA repair cofactors ATMIN and NBS1 are required to suppress T cell activation
J Prochazkova, S Sakaguchi, M Owusu, A Mazouzi, M Wiedner, ...
PLoS genetics 11 (11), e1005645, 2015
Các cơ quan ủy nhiệm: Austrian Science Fund, Cancer Research UK, Wellcome Trust
53BP1‐mediated recruitment of RASSF1A to ribosomal DNA breaks promotes local ATM signaling
S Tsaridou, G Velimezi, F Willenbrock, M Chatzifrangkeskou, W Elsayed, ...
EMBO reports 23 (8), e54483, 2022
Các cơ quan ủy nhiệm: German Research Foundation, European Commission
53BP1 mediated recruitment of RASSF1A at ribosomal DNA breaks facilitates local ATM signal amplification
S Tsaridou, G Velimezi, F Willenbrock, M Chatzifrangkeskou, ...
bioRxiv, 2021.11. 17.469058, 2021
Các cơ quan ủy nhiệm: European Commission
Map of synthetic rescue interactions for the Fanconi anemia DNA repair pathway identifies USP48
SP Jackson, G Velimezi, L Robinson-Garcia, F Muñoz-Martínez, ...
Springer Nature, 2018
Các cơ quan ủy nhiệm: Austrian Science Fund, Cancer Research UK, Wellcome Trust, European Commission
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