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Alois Schweighofer
Alois Schweighofer
Affiliazione sconosciuta
Email verificata su univie.ac.at
Titolo
Citata da
Citata da
Anno
Antagonistic regulation of PIN phosphorylation by PP2A and PINOID directs auxin flux
M Michniewicz, MK Zago, L Abas, D Weijers, A Schweighofer, I Meskiene, ...
Cell 130 (6), 1044-1056, 2007
7952007
Plant PP2C phosphatases: emerging functions in stress signaling
A Schweighofer, H Hirt, I Meskiene
Trends in plant science 9 (5), 236-243, 2004
7952004
The PP2C-Type Phosphatase AP2C1, Which Negatively Regulates MPK4 and MPK6, Modulates Innate Immunity, Jasmonic Acid, and Ethylene Levels in Arabidopsis
A Schweighofer, V Kazanaviciute, E Scheikl, M Teige, R Doczi, H Hirt, ...
The Plant Cell 19 (7), 2213-2224, 2007
3792007
The TPLATE adaptor complex drives clathrin-mediated endocytosis in plants
A Gadeyne, C Sánchez-Rodríguez, S Vanneste, S Di Rubbo, H Zauber, ...
Cell 156 (4), 691-704, 2014
2862014
Type 2C protein phosphatases in plants
S Fuchs, E Grill, I Meskiene, A Schweighofer
The FEBS journal 280 (2), 681-693, 2013
2332013
Stress-induced protein phosphatase 2C is a negative regulator of a mitogen-activated protein kinase
I Meskiene, E Baudouin, A Schweighofer, A Liwosz, C Jonak, ...
Journal of Biological Chemistry 278 (21), 18945-18952, 2003
2092003
Dynamic recruitment of Cdc2 to specific microtubule structures during mitosis
M Weingartner, P Binarova, D Drykova, A Schweighofer, JP David, ...
The Plant Cell 13 (8), 1929-1943, 2001
1162001
MAPK phosphatase AP2C3 induces ectopic proliferation of epidermal cells leading to stomata development in Arabidopsis
J Umbrasaite, A Schweighofer, V Kazanaviciute, Z Magyar, Z Ayatollahi, ...
PloS one 5 (12), e15357, 2010
1072010
Plant resistance against the parasitic nematode Heterodera schachtii is mediated by MPK3 and MPK6 kinases, which are controlled by the MAPK phosphatase …
E Sidonskaya, A Schweighofer, V Shubchynskyy, N Kammerhofer, ...
Journal of experimental botany 67 (1), 107-118, 2016
602016
Salt-induced subcellular kinase relocation and seedling susceptibility caused by overexpression of Medicago SIMKK in Arabidopsis
M Ovečka, T Takáč, G Komis, P Vadovič, S Bekešová, A Doskočilová, ...
Journal of experimental botany 65 (9), 2335-2350, 2014
532014
Protein phosphatase AP2C1 negatively regulates basal resistance and defense responses to Pseudomonas syringae
V Shubchynskyy, J Boniecka, A Schweighofer, J Simulis, ...
Journal of Experimental Botany 68 (5), 1169-1183, 2017
502017
Phosphatases in plants
A Schweighofer, I Meskiene
Plant phosphoproteomics: methods and protocols, 25-46, 2015
362015
Regulation of stress hormones jasmonates and ethylene by MAPK pathways in plants
A Schweighofer, I Meskiene
Molecular BioSystems 4 (8), 799-803, 2008
352008
Dual control of MAPK activities by AP2C1 and MKP1 MAPK phosphatases regulates defence responses in Arabidopsis
Z Ayatollahi, V Kazanaviciute, V Shubchynskyy, K Kvederaviciute, ...
Journal of experimental botany 73 (8), 2369-2384, 2022
222022
Targeting alternative splicing by RNAi: from the differential impact on splice variants to triggering artificial pre-mRNA splicing
A Fuchs, S Riegler, Z Ayatollahi, N Cavallari, LE Giono, BA Nimeth, ...
Nucleic Acids Research 49 (2), 1133-1151, 2021
222021
Protein phosphatases in plant growth signalling pathways
A Schweighofer, I Meskiene
Plant growth signaling, 277-297, 2008
162008
Substrate Analysis of Arabidopsis PP2C-Type Protein Phosphatases
J Umbrasaite, A Schweighofer, I Meskiene
Plant Kinases: Methods and Protocols, 149-161, 2011
152011
Phosphatase activities analyzed by in vivo expressions
A Schweighofer, Z Ayatollahi, I Meskiene
Plant Signal Transduction: Methods and Protocols, 247-260, 2009
72009
Bimolecular fluorescent complementation (BiFC) by MAP kinases and MAPK phosphatases
A Schweighofer, V Shubchynskyy, V Kazanaviciute, A Djamei, I Meskiene
Plant MAP Kinases: Methods and Protocols, 147-158, 2014
62014
Remote control of alternative splicing in roots through TOR kinase
E Petrillo, S Riegler, A Fuchs, L Servi, MA Godoy Herz, MG Kubaczka, ...
BioRxiv, 472126, 2018
32018
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